![Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression | Gut Pathogens | Full Text Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression | Gut Pathogens | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs13099-020-00363-8/MediaObjects/13099_2020_363_Fig1_HTML.png)
Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression | Gut Pathogens | Full Text
1 MATERIALS AND METHODS Cell lines and culture conditions. AGS human gastric cancer cells were directly purchased from the Ameri
Alterations in cellular expression in EBV infected epithelial cell lines and tumors | PLOS Pathogens
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PDF] Unique enhancement of multinuclear giant cell formation in AGS cell line infected with Helicobacter pylori | Semantic Scholar
![Altered states: Involvement of phosphorylated CagA in the induction of host cellular growth changes by Helicobacter pylori | PNAS Altered states: Involvement of phosphorylated CagA in the induction of host cellular growth changes by Helicobacter pylori | PNAS](https://www.pnas.org/cms/10.1073/pnas.96.25.14559/asset/0f9e0f6e-2a99-4ede-bdfd-6fe298fcdb82/assets/graphic/pq2594423004.jpeg)
Altered states: Involvement of phosphorylated CagA in the induction of host cellular growth changes by Helicobacter pylori | PNAS
Multicellular Human Gastric-Cancer Spheroids Mimic the Glycosylation Phenotype of Gastric Carcinomas
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Pathogens | Free Full-Text | Cortactin Promotes Effective AGS Cell Scattering by Helicobacter pylori CagA, but Not Cellular Vacuolization and Apoptosis Induced by the Vacuolating Cytotoxin VacA
![Biomolecules | Free Full-Text | Benzyl Isothiocyanate Induces Apoptosis via Reactive Oxygen Species-Initiated Mitochondrial Dysfunction and DR4 and DR5 Death Receptor Activation in Gastric Adenocarcinoma Cells Biomolecules | Free Full-Text | Benzyl Isothiocyanate Induces Apoptosis via Reactive Oxygen Species-Initiated Mitochondrial Dysfunction and DR4 and DR5 Death Receptor Activation in Gastric Adenocarcinoma Cells](https://www.mdpi.com/biomolecules/biomolecules-09-00839/article_deploy/html/images/biomolecules-09-00839-g001-550.jpg)
Biomolecules | Free Full-Text | Benzyl Isothiocyanate Induces Apoptosis via Reactive Oxygen Species-Initiated Mitochondrial Dysfunction and DR4 and DR5 Death Receptor Activation in Gastric Adenocarcinoma Cells
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Morphological changes of apoptosis and cytotoxic effects induced by Caffeic acid phenethyl ester in AGS human gastric cancer cell line | Semantic Scholar
![Flavonoids Isolated from Korea Citrus aurantium L. Induce G2/M Phase Arrest and Apoptosis in Human Gastric Cancer AGS Cells Flavonoids Isolated from Korea Citrus aurantium L. Induce G2/M Phase Arrest and Apoptosis in Human Gastric Cancer AGS Cells](https://static-02.hindawi.com/articles/ecam/volume-2012/515901/figures/515901.fig.002b.jpg)
Flavonoids Isolated from Korea Citrus aurantium L. Induce G2/M Phase Arrest and Apoptosis in Human Gastric Cancer AGS Cells
![Synergistic anticancer properties of docosahexaenoic acid and 5-fluorouracil through interference with energy metabolism and cell cycle arrest in human gastric cancer cell line AGS cells Synergistic anticancer properties of docosahexaenoic acid and 5-fluorouracil through interference with energy metabolism and cell cycle arrest in human gastric cancer cell line AGS cells](https://f6publishing.blob.core.windows.net/39f2f3f1-7b98-4410-a016-720bec1e9a65/WJG-22-2971-g002.jpg)
Synergistic anticancer properties of docosahexaenoic acid and 5-fluorouracil through interference with energy metabolism and cell cycle arrest in human gastric cancer cell line AGS cells
![Helicobacter pylori CagA Induces AGS Cell Elongation through a Cell Retraction Defect That Is Independent of Cdc42, Rac1, and Arp2/3 | Infection and Immunity Helicobacter pylori CagA Induces AGS Cell Elongation through a Cell Retraction Defect That Is Independent of Cdc42, Rac1, and Arp2/3 | Infection and Immunity](https://journals.asm.org/cms/10.1128/IAI.01702-06/asset/0af5e039-535c-4179-96d0-d715d84283d4/assets/graphic/zii0030765300006.jpeg)
Helicobacter pylori CagA Induces AGS Cell Elongation through a Cell Retraction Defect That Is Independent of Cdc42, Rac1, and Arp2/3 | Infection and Immunity
![BP‑1‑102 exerts an antitumor effect on the AGS human gastric cancer cell line through modulating the STAT3 and MAPK signaling pathways BP‑1‑102 exerts an antitumor effect on the AGS human gastric cancer cell line through modulating the STAT3 and MAPK signaling pathways](https://www.spandidos-publications.com/article_images/mmr/19/4/MMR-19-04-2698-g03.jpg)
BP‑1‑102 exerts an antitumor effect on the AGS human gastric cancer cell line through modulating the STAT3 and MAPK signaling pathways
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In vitro and in vivo studies on antitumor effects of gossypol on human stomach adenocarcinoma (AGS) cell line and MNNG induced experimental gastric cancer - ScienceDirect
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The effect of paclitaxel on apoptosis, autophagy and mitotic catastrophe in AGS cells | Scientific Reports
![Identification of different gene expressions between diffuse- and intestinal-type spheroid-forming gastric cancer cells | SpringerLink Identification of different gene expressions between diffuse- and intestinal-type spheroid-forming gastric cancer cells | SpringerLink](https://media.springernature.com/lw685/springer-static/image/art%3A10.1007%2Fs10120-019-00935-x/MediaObjects/10120_2019_935_Fig2_HTML.png)
Identification of different gene expressions between diffuse- and intestinal-type spheroid-forming gastric cancer cells | SpringerLink
![Live-cell micrographs of H. pylori-infected AGS cells. AGS cells were... | Download Scientific Diagram Live-cell micrographs of H. pylori-infected AGS cells. AGS cells were... | Download Scientific Diagram](https://www.researchgate.net/publication/279302996/figure/fig2/AS:340507206144013@1458194652842/Live-cell-micrographs-of-H-pylori-infected-AGS-cells-AGS-cells-were-infected-with-H.png)
Live-cell micrographs of H. pylori-infected AGS cells. AGS cells were... | Download Scientific Diagram
![Interleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells | BMC Microbiology | Full Text Interleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells | BMC Microbiology | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2F1471-2180-12-9/MediaObjects/12866_2011_Article_1580_Fig1_HTML.jpg)
Interleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells | BMC Microbiology | Full Text
1 MATERIALS AND METHODS Cell lines and culture conditions. AGS human gastric cancer cells were directly purchased from the Ameri
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Figure 3 | CDK-associated Cullin 1 can promote cell proliferation and inhibit cisplatin-induced apoptosis in the AGS gastric cancer cell line | SpringerLink
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